Strictly Science: Long SARS-CoV-2 Something

Sorting out cryptic messages of the latest study...

A very brief recap of the official line first. By now, we have been told innumerous times that:

• The Covid mRNA “vaccines” are safe (don’t ask how safe). and effective.

• The mRNA gets degraded very fast (don’t ask how fast) in the bodies of so jabbed.

• And only while the mRNA is there for a brief time (don’t ask how brief) in your body, it churns out the S spike proteins that mimic the virus (don’t ask if they cause any disease) and stimulate the immune system to produce the coveted B cells that churn out S spike antibodies.

• The S spikes thus produced don’t last for long (don’t ask for how long) and get degraded rapidly (don’t ask how rapidly).

  The jab mRNA never enters the cell nuclei and never imbeds into your DNA (unless it does, as it turns out, here and here).

• The S spike antibodies latch on to any virus that might happen to invade you. Thus these antibodies prevent the SARS-CoV-2 viruses from entering your cells, thus conferring “protection against the severe disease” (although initially it was claimed to confer immunity) .

You can see that the last statement of faith doesn’t add up: the viruses get covered with antibodies and can’t replicate yet they still replicate and get passed on.

But that isn’t the subject of this post. The subject of this post is the study that tested study subjects that have been diagnosed with acute SARS-CoV-2 infection, for any lingering health issues they might have up to 910 days later. “Tissue-based T cell activation and viral RNA persist for up to 2 years after SARS-CoV-2 infection” (Science, 2024.07.03):

We performed whole-body positron emission tomography imaging in a well-characterized cohort of 24 participants at time points ranging from 27 to 910 days after acute SARS-CoV-2 infection using the radiopharmaceutical agent [18F]F-AraG, a selective tracer that allows for anatomical quantitation of activated T lymphocytes. Tracer uptake in the postacute COVID-19 group, which included those with and without continuing symptoms, was higher compared with prepandemic controls in many regions, including the brain stem, spinal cord, bone marrow, nasopharyngeal and hilar lymphoid tissue, cardiopulmonary tissues, and gut wall. T cell activation in the spinal cord and gut wall was associated with the presence of LC symptoms. In addition, tracer uptake in lung tissue was higher in those with persistent pulmonary symptoms specifically. Increased T cell activation in these tissues was also observed in many individuals without LC.

This is what the PET scans showed:

Figure S1. Maximum intensity projections (MIP; coronal views of 3-dimensional reconstructions) show PET data for each participant and pre-COVID-19 Control. (A and B) MIPs are shown for all cases (A) and pre-pandemic control participants (B). M, Male; F, Female; SUV, standardized uptake value.

The processed statistics for the above:

Figure S3. Increased [18F]F-AraG SUVmax and SUVmean uptake in right hilar ROIs in male sex assigned at birth post-acute COVID-19. (A and B) Bars represent mean SUVmax (A) and SUVmean (B); error bars represent 95% confidence interval. Adjusted P values <0.05 and <0.001 represented by * and **, respectively from two-sided non-parametric Kruskal–Wallis tests using a Benjamini-Hochberg adjustment for false discovery rates across multiple comparisons (q value = adjusted P value). Given lack of power to compare cases versus controls grouped on sex, statistical analyses were only performed between female and male post-acute COVID-19 participants. All data points are shown.

I hear you cry: ”What about their vaccination statuses?!” As the full study is not available, there in no word about it. Yet! I have found the study preprint from Jul. 31, 2023. As it turns out, all but one (participant No. 6) study subjects have been “vaccinated” (probably multiple times), with the most recent jab occurring before (10) or after (13) the diagnosed infection:

So much for the jab protecting against Long Covid, before or after the jab. One lucky study subject even has been jabbed 6 days before the PET scan, only two months after being diagnosed with Covid (“To minimize the impact of vaccination on T cell activation, PET imaging was performed greater than 60 days from any vaccine dose (SARS-CoV-2 or otherwise), but one participant received a SARS-CoV-2 booster vaccine dose 6 days prior to imaging without notifying the study team.“)

Yet, the best is yet to follow:

This study provides evidence for ongoing immune responses in tissues, a potentially important source of inflammation observed in peripheral blood. …This is consistent with a prior autopsy study, which demonstrated the presence of SARS-CoV-2 spike RNA and protein in the spinal cord and basal ganglia in two individuals that died during the post-acute phase following COVID-19 (65 and 230 days post infection).

We observed that T cell activation in spinal cord and gut wall was associated with the presence of Long COVID symptoms. In addition, tracer uptake in lung tissue was higher in those with persistent pulmonary symptoms. Notably, increased T cell activation in these tissues was also observed in many individuals without Long COVID.

Intestinal biopsies in a subset of post-acute COVID participants show evidence of persistent SARS-CoV-2 spike RNA in rectal tissue. SARS-CoV-2 RNA was readily detected in multiple cells from all three tissue regions surveyed from all but one individual (participant 15) who had rare RNA+ cells detected in only one of three gut tissue regions 645 days following initial infection.

Which is telling us that the study looked strictly at the S spike protein and the S spike RNA. It didn’t include the nucleocapcid protein or RNA. Which is telling us that none were to be found. Yet, the S spike protein and RNA abound. Isn’t that something!

And the T cell activation is speaking volumes too:

T cell activation requires extracellular stimulatory signals that are mainly mediated by T cell receptor (TCR) complexes. The TCR recognizes antigens on major histocompatibility complex molecules with the cooperation of CD4 or CD8 coreceptors. After recognition, TCR-induced signaling cascades that propagate signals via various molecules and second messengers are induced.

This means that the body cells present these S spike proteins on their surfaces once found inside the cells. Meaning that there is a constant supply of S spike proteins inside the cells of those with Long Covid. Hence the ongoing inflammation, a.k.a. the the killing of the cells presenting the S spike on their surfaces by one’s immune system.

Of course, the study authors don’t put it that bluntly: they blame the disregulation of the T-cell responses for the ongoing inflammation. Yet, the authors contradict themselves by linking the presence of S spike proteins and RNA in the affected tissues with the inflammation.

Maybe they are trying to tell us something, by playing stupid?

You can read the mainstream interpretation of this study here: “Long COVID uncharacteristic immune cell activity and SARS-CoV-2 RNA found in the gut two years after infection” (Medical Xpress, 2024.07.08):

The research team suggests long COVID is a real biological illness and that the SARS-CoV-2 virus can persist in the body much longer than has been thought.

Despite or thank to all the jabs?