Long Covid [Vax] Conditions

The gaslighting continues in earnest.

According to the CDC’s “Long COVID or Post-COVID Conditions” (CDC, July 2023):

Long COVID is a wide range of new, returning, or ongoing health problems that people experience after being infected with the virus that causes COVID-19.

Most people with Long COVID experienced symptoms days after first learning they had COVID-19, but some people who later experienced Long COVID did not know when they got infected.

So, the CDC knows what it isn’t (emphatically not a Covid “vaccine” injury), yet they say there are no tests to say what it is, and they have no clue either:

There is no test that determines if your symptoms or condition is due to COVID-19. Long COVID is not one illness. Your healthcare provider considers a diagnosis of Long COVID based on your health history, including if you had a diagnosis of COVID-19 either by a positive test or by symptoms or exposure, as well as based on a health examination.

Well, let me rain on this charade. “Acute blood biomarker profiles predict cognitive deficits 6 and 12 months after COVID-19 hospitalization” (Nature, 2023.08.31):

In this prospective cohort study of 1,837 adults hospitalized with COVID-19, we identified two distinct biomarker profiles measured during the acute admission, which predict cognitive outcomes 6 and 12 months after COVID-19.

A first profile links elevated fibrinogen relative to C-reactive protein with both objective and subjective cognitive deficits.

A second profile links elevated D-dimer relative to C-reactive protein with subjective cognitive deficits and occupational impact. This second profile was mediated by fatigue and shortness of breath.

In addition, “SARS-CoV-2 reservoir in post-acute sequelae of COVID-19 (PASC)”(Nature Immunology, 2023.09.04):

Millions of people are suffering from Long COVID or post-acute sequelae of COVID-19 (PASC). Several biological factors have emerged as potential drivers of PASC pathology. Some individuals with PASC may not fully clear the coronavirus SARS-CoV-2 after acute infection. Instead, replicating virus and/or viral RNA—potentially capable of being translated to produce viral proteins—persist in tissue as a ‘reservoir’. This reservoir could modulate host immune responses or release viral proteins into the circulation. Here we review studies that have identified SARS-CoV-2 RNA/protein or immune responses indicative of a SARS-CoV-2 reservoir in PASC samples.

Adding 2 and 2 together, (a) the PASC is presented as micro blood clots in the brain and/or lungs (which simple off-the-shelf blood tests can identify), and (b) these micro-clots are being caused by persistent “viral proteins” being released into the blood stream.

And we all know the role of S spike SARS-CoV-2 protein in the blood clot formation, don’t we? Just as a recap, “SARS-CoV-2 spike protein S1 induces fibrin(ogen) resistant to fibrinolysis: Implications for microclot formation in COVID-19”(Medrxiv, 2021.03.05):

Severe acute respiratory syndrome coronavirus 2 (SARS-Cov-2)-induced infection, the cause of coronavirus disease 2019 (COVID-19), is characterized by unprecedented clinical pathologies. One of the most important pathologies, is hypercoagulation and microclots in the lungs of patients. Here we study the effect of isolated SARS-CoV-2 spike protein S1 subunit as potential inflammagen sui generis.

Using platelet poor plasma (PPP), we show that spike protein may interfere with blood flow. Mass spectrometry also showed that when spike protein S1 is added to healthy PPP, it results in structural changes to β and γ fibrin(ogen), complement 3, and prothrombin. These proteins were substantially resistant to trypsinization, in the presence of spike protein S1. Here we suggest that, in part, the presence of spike protein in circulation may contribute to the hypercoagulation in COVID-19 positive patients and may cause substantial impairment of fibrinolysis.

And we also know that the amount of “benign” S spike produced in the “vaccinee” is orders of magnitude higher and stays in the “vaccinee” longer than during the “regular” Covid infection (e.g., see my posts “Does mRNA in Jabs Really Produce S Spike from SARS-CoV-2?” from November 2021 and “SARS-CoV-2 S Spike To Centre Stage” from June 2022). Therefore, although some Covid-recovered may develop “Long Covid”, the much higher chances are of developing a “Long Covid Jab” after taking one or more “vaccine” injections.

I don’t have a direct proof of that with a well-designed study tackling this exact question head-on. Yet, here’s my anecdote as an illustration. One evening this summer I was descending from a steep hill that is crowned by an ancient chapel, somewhere in Europe. Almost at the top, an out-of-breath middle-aged woman was resting her hand on a stone wall, catching her breath. I have cheered her on by saying she’s almost there at the top. And then the woman explained that due to Long Covid she has been extremely tired and short of breath for the last year, also with a brain fog. And although she’s from that town, she can hardly climb this hill now. I suggested some supplements. Then the woman, with tears in her eyes, confessed: “I know I developed this condition after a Covid vaccine! I only took one, and now I have to deal with this…” Obviously, her physicians were telling her she was insane to blame the “safe and effective” vaccines for her predicament - must be some genetic hereditary condition of hers. Or the Long Covid. No other possibilities. Now she distrusts the medical profession, but being in the perfect Covid information vacuum of an “advanced” European “democracy”, she could not find the right, actionable information. All I could do was to refer her to the FLCCC post-jab protocol. Hopefully she will find the clot-fighting supplements useful and effective and will recover soon.