The claim: COVID-19 vaccines 'speed up the aging process'. "Those who are fully vaccinated, will age faster than those who are not vaccinated and possess age related diseases way before their time."
Our rating: False. There is no evidence the COVID-19 vaccines speed up the aging process, an expert told USA TODAY. The same can't be said for the virus it helps prevent, however: Some researchers have found links between severe cases of COVID-19 and signs of accelerated aging.
Isn’t it amazing how the fact checkers and experts want to have it both ways, on a single breath? If we were to trust these “experts”, severe Covid does cause the accelerated aging, while the jabs that flood the injected with 10-100 times more of the toxic S spike [that causes Covid in the first place] (my post “SARS-CoV-2 S Spike To Centre Stage”, 2022.06.06) ) for months on end, don’t? Stretches my credulity. Time for a closer look!
First for the fact checkers’ argument that ZJab doesn’t cause aging. Their argumentation is so simple that it fits into half a sentence: because ZJab prevents the severe Covid disease. Q.E.D. Although somehow more people died after receiving the jab than prior, if we compare the official Covid death stats in 2021 vs 2022 in the highly vaccinated countries like Canada, for example, so this conjecture is highly questionable (my post “What If S Spike-Targeting Jabs Never Worked, Like At All?”, 2022.06.02).
As for their argument that severe Covid does cause accelerated aging, to their credit, they provide the references to back it up, which is not a small feat for a fact checker:
“We observed that gene expression in the brain tissue of patients who died of COVID-19 closely resembled that of uninfected individuals 71 years old or older,” said co-first author Jonathan Lee, a postdoctoral research fellow at BIDMC and Harvard Medical School. “Genes that were upregulated in aging were upregulated in the context of severe COVID-19; likewise, genes downregulated in aging were also downregulated in severe COVID-19.”
“While we did not find evidence that the SARS-CoV-2 virus was present in the brain tissue at the time of death, we discovered inflammatory patterns associated with COVID-19. This suggests that this inflammation may contribute to the aging-like effects observed in the brains of patients with COVID-19 and long COVID,” Lee added. “Given these findings, we advocate for neurological follow-up of recovered COVID-19 patients,” said senior and co-corresponding author Frank Slack, director of the Institute for RNA Medicine at BIDMC and the Shields Warren Mallinckrodt Professor of Medical Research at Harvard Medical School.
“Taken together, our data suggest that COVID-19-induced TNF and type I/II interferons may lead to significant deteriorating effects in the brainin the absence of SARS-CoV-2 neuroinvasion.”
A key challenge after the COVID-19 pandemic will be managing the long-term sequelae for the millions of individuals who recover from the disease. Based on the available evidence, our hypothesis is that the SARS-CoV-2 pandemic and its long-term complications will lead to premature aging (in terms of health) of many people in the world.
In this study, DNA methylation of the blood samples from 232 healthy individuals and 413 COVID-19 patients is profiled using EPIC methylation array. Epigenetic ages of each individual are determined by applying epigenetic clocks (Hannum, Horvath, PhenoAge, skinHorvath and GrimAge clocks) and telomere length estimator to the methylation profile of the individual. We defined epigenetic age acceleration for each case by comparing the individual’s epigenetic age with chronological age to assess whether the accelerated or dysfunctional epigenetic aging is associated with SARS-CoV-2 infection and the severity of COVID-19 syndrome.
Our analysis of epigenetic clocks and telomere shortening indicates an increasing age acceleration at the initial phases of COVID-19 and this could be partly reversed at later phases. We speculated that COVID-19 syndrome might accelerate epigenetic aging in SARS-CoV-2-infected patients based on these findings. Our longitudinal [AO: i.e., following through time]DNA methylation profiling analysis indicates that the influence of COVID-19 syndrome on epigenetic aging in peripheral blood could be reversed in some patients.
a Acceleration of DNAm aging (five epigenetic clocks) and telomere attrition of the blood samples collected from uninfected control and patients at each clinical disease phase. b Dynamic change of epigenetic age acceleration in each individual across COVID-19 disease phases. The p value for each t-test is shown above the corresponding line. In the box plots, the lower and upper hinges indicate the 25th and 75th percentiles, and the black line within the box marks the median value. The whiskers extend from the hinges to the largest and smallest values no further than 1.5 × inter-quartile range from the hinges, and the points beyond the end of whiskers indicate outliers. Source data are provided as a Source Data file.
So, in those that have died with Covid, we see dramatic inflammatory markers and “aging” in their brains, as compared to the dead without Covid (1st study). Also, we see the age clocks dialed forward in the serum of those that are sick with Covid, then being dialed back as the person recovers (the 3rd study). Nothing of substance in the 2nd study, which is actually a compendium of anecdotes.
But, there is nothing in the referenced sources that would prove that ZJab isn’t doing the same or worse on the jabbed - it just wasn’t the subject of these studies. So, it’s a leap of blind faith to declare that ZJab isn’t causing similar or worse, and also chronic, aging on the part of the fact checkers.
A quick search of Substack yielded this post claiming to sum up our knowledge, head-on, on the aging issues related to Covid:
To create a more fine-grained analysis of the cellular gene expression states in each tissue, we used the cell deconvolution multiple signal classification algorithm (MuSiC) on each tissue’s RNA-seq data (see STAR Methods). The MuSiC results showed distinct disruptions of the transcriptional programs for each type of tissue in the COVID-19 patients and in the gain or loss of cell types (Figure 2B). Consistent with previous reports, the lung showed a loss of the capillary intermediate cells and alveolar epithelial cell types.10,11 Strikingly, we also found decreases in the major cell types in each organ type, suggesting a systemic disruption of the COVID-19 response. The kidney and liver showed a loss of proximal tubule in the kidney and of hepatocyte marker expressions in the liver but an increase in T cells in both organs. Furthermore, the heart showed a near-complete loss of the cell signatures for cardiomyocytes in both SARS-CoV-2 high and low viral loads (Figures 2B and and3A),3A), despite no obvious gross or histologic changes in the heart (Figure 3B). This observation extends from previously reported cardiovascular involvements in COVID-19 and further defines the SARS-CoV-2-specific transcript and cellular changes in lung as well as other organs such as heart, liver, and kidney.
(A) The pathways that show significant differences in all or the majority of the five tissue types are shown, with statistical significance from GSEA testing across five kinds of tissue (colors in legend), including heart (n = 41), kidney (n = 27), liver (n = 40), lung (n = 40), and lymph node (n = 27). The x axis shows the normalized enrichment score for COVID-19-positive versus control, SARS-CoV-2 high or low versus control, and SARS-CoV-2 high versus Low comparisons.
It looks like yet another study the fact checkers could have used to support their argument. We see that all 5 major organ tissues are affected by Covid-19, but why?
In 13/13 brains from fatal COVID-19, pseudovirions (spike, envelope, and membrane proteins without viral RNA) were present in the endothelia of microvessels ranging from 0 to 14 positive cells/200× field (mean 4.3). The pseudovirions strongly co-localized with caspase-3, ACE2, IL6, TNFα, and C5b-9.
This harks back to the 1st study cited by the fact checkers, that claimed no viral RNA in the brains of the Covid dead but “we discovered inflammatory patterns associated with COVID-19”. But this one goes further and pinpoints the brain inflammation patterns to the endothelia of the microvessels. To test which pseudovirions cause this inflammation, the researchers injected mice with different components of the virus and determined that S1 subunit of the S spike is the culprit:
Tail vein injection of the full length S1 spike subunit in mice led to neurologic signs (increased thirst, stressed behavior) not evident in those injected with the S2 subunit. The S1 subunit localized to the endothelia of microvessels in the mice brain and showed co-localization with caspase-3, ACE2, IL6, TNFα, and C5b-9.
Bingo! Exactly same as in the brains of the Covid-19 dead. In addition to inflammation, other markers have pointed to neural disfunction caused by the S1 subunit of the S spike:
In addition, Glutamate NMDA receptor 2 (NMDAR2) and neuronal nitric oxide synthetase (nNOS) have each been implicated as markers of neuronal dysfunction. NMDAR2 was not detected in the normal brain tissues. It was expressed in the COVID-19 brains where it was evident in neurons adjacent to SARS-CoV-2 positive microvessels (Table 1 and Fig. 2). Similarly, the percentage of neurons with nNOS was significantly greater (p < 0.001) in the COVID-19 brain tissues compared to the controls.
Here’s your the mental decline that comes from the abundance of the S spike in the brain.
And now let me recap the information we have about the biodistribution, pharmacokinetics, amount and persistence of the S spike as a result of Covid-19 infection versus Covid-19 vaccination with an mRNA-based Covid-19 “vaccine” (the viral vector-based jabs like J&J and AstraZeneca seem to be even worse as those have been withdrawn in a short order for causing more severe adverse effects on the jab recipients than the mRNA jabs):
The lipid nanoparticle (LNP) wrapped mRNA was originally designed to penetrate every barrier (including the blood-brain barrier, or BBB) and every cell membrane, to work as an mRNA delivery vehicle, or a bullet, for cancer treatments. And it works as such in the Covid jabs as well - the Pfizer submission to the Japanese received under FOIA request documents just that (“A Moratorium on mRNA 'Vaccines' is Needed”, Byram Briddle, 2022.04.21) - within 48 hours and if you are lucky, a mere 25% of the injected material remains at the injection site, the rest is distributed throughout the other organs, where the LNPs enter the cells and the mRNA within is released and starts churning out loads of S spikes. That included the brain.
The quantity of S spike produced by the “vaccination” is 10-100 times (orders of magnitude) higher than the amount of spike produced in the course of a Covid infection that usually lasts for up to two weeks — contrast that with 4 months after each “vaccination” (again, in my post “SARS-CoV-2 S Spike To Centre Stage”, 2022.06.06).
Taking all of the above into account, it’s safe to conclude that mRNA jabs are causing much grater and much more lasting aging effect following every “vaccination” as does even the most severe Covid, unless it goes chronic in the very rare immune-compromised cases.
My rating:Correct. It is indisputable that those who are vaccinated will age faster following every “vaccination” event for at least 4 months on end. We have the concrete solid evidence for that. The question remains open as to how fast and how much the jab recipients will be able to recover once they stop “boosting” their way into the afterlife.
As an indirect corroboration of this, I and many others have observed the widespread senile (one might say asinine) driving patterns on the major highways during and shortly after the major jabbination campaigns back in 2021. The fact that this phenomenon has mostly went away by now gives hope that the brains do eventually recover given enough break from the mRNA S spike jabbination.
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Wow! Blood Brain Barrier can be denoted as BBB - another term for “Build Back Better” as part of the WEF’s campaign for “The Great Reset”.
One could say that BBB is a Janus-faced word which is code for GENOCIDE 1) via crossing the blood-brain barrier thru use of a neuro--physiological attack (the shots) which incapacitates & ultimately destroys ppl’s minds & 2) via an attack thru a reset of society which limits all resources bringing the population into a subsistence existence leading to death by starvation.
Wow! Blood Brain Barrier can be denoted as BBB - another term for “Build Back Better” as part of the WEF’s campaign for “The Great Reset”.
One could say that BBB is a Janus-faced word which is code for GENOCIDE 1) via crossing the blood-brain barrier thru use of a neuro--physiological attack (the shots) which incapacitates & ultimately destroys ppl’s minds & 2) via an attack thru a reset of society which limits all resources bringing the population into a subsistence existence leading to death by starvation.
Excellent. Exquisite logic.